Background and objective Post-activation potentiation (PAP) refers to the improvement of explosive power in a short period of time, but it needs to be induced by high-intensity resistance training. It has been reported that blood flow restriction combined with low-intensity resistance training (BFR-RT) can produce similar effects on skeletal muscle as high-intensity resistance training. However, the number of studies on the induction of PAP by BFR-RT is limited. Therefore, this study explored the effect of BFR-RT on PAP by pooling relevant studies. News PubMed, Web of Science, ScienceDirect, Scopus, EBSCO, and CNKI (Chinese database) were searched for randomized controlled trial (RCT) of the effect of BFR-RT on PAP from the time of database construction until September 20, 2022. The effects on outcomes were expressed as standardized mean difference (SMD) along with 95% confidence intervals (CI). Thirteen articles with a total of 51 studies and 195 subjects were included in the Revman5.4 software for meta-analysis. BFR-RT significantly improves jump height (JH) (SMD = 0.49, 95%CI [0.31, 0.66]), power output (PO) (SMD = 0.49, 95%CI [0.30, 0.68]), rate of force development (RFD) (SMD = 0.60, 95%CI [0.32, 0.88]), reactive strength index (RSI) (SMD = 0.46, 95% CI [0.02, 0.90]) compared to the control group. Arterial occlusion pressure (AOP) = 60% reached maximum effect (SMD = 0.66, 95%CI [0.48, 0.83]) and was statistically significant. Resistance training intensity = 30% 1 - repetition maximum (1RM) reached the maximum effect (SMD = 0.54, 95%CI [0.38, 0.71]) and was statistically significant. Conclusion This meta-analysis found that BFR-RT could induce PAP. In addition, a combination of 60% ~ 80% AOP and 20% ~ 30% 1RM could induce greater PAP.
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