The aim of this article is to assess the role of lactate in metabolic acidosis, muscle fatigue and exercise-induced adaptation. The view of the role of lactate has changed essentially during the last 30 years. Lactate is not considered as the main cause of fatigue as it had been previously, but instead as a marker of anaerobic catabolism of glucose. The lactate is produced also in aerobic conditions and is shuttled between cells. It serves as a major energy source, the major gluconeogenic precursor, as well as asignalling molecule that mediates exercise-induced adaptations.
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