The biology and medicine of rowing are briefly reviewed. Effort in a 2000-m race is about 70% aerobic. Because the boat (and in some instances a cox) must be propelled, successful competitors are very tall, with a large lean mass and aerobic power. Large hearts may lead to erroneous diagnoses of a cardiomyopathy. Large respiratory minute volumes must be developed by chest muscles that are also involved in rowing. The vital capacity is typically large, and breathing becomes entrained. Expiration cannot be slowed relative to inspiration (as normally occurs at high rates of ventilation) and the limiting flow velocity may be reached, with the potential for airway collapse. Performance is strongly related to the power output at the `anaerobic threshold', and lactate measures provide a guide to an appropriate intensity of endurance training. Peak blood lactate levels are higher in males (commonly 11–19 mmol&12;·&12;l −1 and occasionally as high as 25 mmol&12;·&12;l −1) than in females (9–11 mmol&12;·&12;l −1), probably because males have a greater muscle mass in relation to blood volume. The skeletal muscles are predominantly slow twitch in type, developing an unusual force and power at low contraction velocities. Many rowers have a suboptimal diet, eating excessive amounts of fat. Lightweight rowers also have problems of weight cycling. Aerobic power and muscle endurance often change by 10% over the season, but such fluctuations can be largely avoided by a well-designed winter training programme. Injuries include back and knee problems, tenosynovitis of the wrist and, since the introduction of large blades, fractures of the costae.
© Copyright 1998 Journal of Sports Sciences. Taylor & Francis. All rights reserved.